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Gastroenterology · USMLE Step 1

USMLE Gastroenterology: Hepatobiliary and Pancreatic Disease

Cirrhosis complications, liver function test patterns, and pancreatitis mechanisms tested on USMLE Step 1, deeper than the general GI overview deck.

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What causes esophageal varices in cirrhosis?
Portal hypertension forces blood through portosystemic collaterals, including the left gastric vein into the esophageal venous plexus
What two factors drive ascites formation in cirrhosis?
Portal hypertension (increased hydrostatic pressure) plus hypoalbuminemia (decreased oncotic pressure)
A serum-ascites albumin gradient (SAAG) of 1.1 g/dL or greater suggests what category of cause?
Portal hypertension as the cause of the ascites
A SAAG less than 1.1 g/dL suggests what category of cause for ascites?
A non-portal-hypertensive cause, such as peritoneal carcinomatosis or infection
What is the most common causative organism of spontaneous bacterial peritonitis (SBP)?
Escherichia coli
What ascitic fluid PMN count is diagnostic of spontaneous bacterial peritonitis?
Greater than or equal to 250 cells/mm3
What molecule is thought to accumulate and drive hepatic encephalopathy in cirrhosis?
Ammonia, due to impaired hepatic clearance and portosystemic shunting
What is the first-line treatment for hepatic encephalopathy?
Lactulose
What is the underlying mechanism of hepatorenal syndrome?
Splanchnic vasodilation from portal hypertension leads to effective arterial underfilling and reflex renal vasoconstriction
What is the underlying mechanism of hepatopulmonary syndrome?
Intrapulmonary vascular dilation causes ventilation-perfusion mismatch and shunting, leading to hypoxemia
What causes the caput medusae sign in cirrhosis?
A recanalized paraumbilical vein shunts portal blood to the abdominal wall veins around the umbilicus
Portal hypertension can cause internal hemorrhoids by forming a shunt between which two venous systems?
The superior rectal vein (portal) and the middle/inferior rectal veins (systemic)
Why does cirrhosis commonly cause gynecomastia in men?
The damaged liver metabolizes estrogen less effectively, so estrogen levels rise relative to androgens
Why is PT/INR often the earliest lab marker to rise in acute liver synthetic dysfunction?
Clotting factor VII has the shortest half-life of the liver-made coagulation factors, so its drop shows up quickly
An AST to ALT ratio greater than 2 to 1 is classically associated with what condition?
Alcoholic liver disease

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