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INBDE · INBDE

INBDE Pharmacology for Dentistry

Local anesthetics, analgesics, and antibiotics commonly prescribed in dental practice tested on the INBDE.

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What is the mechanism of action of local anesthetics?
They block voltage-gated sodium channels in the nerve membrane, preventing depolarization.
What are the two chemical classes of local anesthetics?
Esters and amides.
Where are ester-type local anesthetics metabolized?
In the plasma, by pseudocholinesterase (hydrolysis).
Where are amide-type local anesthetics metabolized?
In the liver, by microsomal enzymes.
Name a commonly used ester local anesthetic in dentistry.
Procaine (or benzocaine).
Name a commonly used amide local anesthetic in dentistry.
Lidocaine.
Why are true allergic reactions more common with ester anesthetics than amides?
Ester metabolism produces PABA, a known allergen.
Why is a vasoconstrictor added to a local anesthetic solution?
To prolong the duration of action and reduce systemic absorption and toxicity.
What is the most commonly used vasoconstrictor in dental local anesthetics?
Epinephrine.
Which local anesthetic is most associated with causing methemoglobinemia?
Prilocaine.
How does a lower pKa affect a local anesthetic's onset of action?
It produces a faster onset, since more drug exists in the un-ionized, lipid-soluble form at tissue pH.
What structural feature makes articaine unique among amide anesthetics?
It has a thiophene ring and an additional ester linkage, allowing partial metabolism in the plasma.
What is an early sign of local anesthetic systemic toxicity?
CNS excitation, such as circumoral numbness, tinnitus, or lightheadedness.
What is the maximum recommended epinephrine dose for a patient with significant cardiovascular disease?
0.04 mg, about two cartridges of 1:100,000 epinephrine.
What is the mechanism of action of NSAIDs?
They inhibit cyclooxygenase (COX) enzymes, decreasing prostaglandin synthesis.

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